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(V103) |
Volume overload-triggered myocardial remodeling occurs in the absence of calcium/calmodulin-dependent protein kinase type II delta in mice |
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B. A. Mohamed, M. Elkenani, J. Jakubiczka-Smorag, S. Koszewa, J. Backs, G. Hasenfuß, K. Toischer, für die Studiengruppe: DZHK (Göttingen, Heidelberg) |
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Diskussion |
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(V104) |
The novel role of calmodulin regulation on myofilament function: calmodulin-myosin interaction discovered |
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V. Sequeira, N. Hamdani, E. Bertero, S. Conijn, C. Ottenheijm, J. van der Velden, C. Maack, C. Johnson (Würzburg, Bochum; Amsterdam, NL; Nashville, US) |
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Diskussion |
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(V105) |
Oxytocin blocks IKs and IK1 and exerts synergistic harmful cardiac repolarization prolonging effects in context of IKr-blocker induced drug-induced LQTS |
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I. Bodi, P. Kreifels, G. Franke, S. Perez-Feliz, A. Castiglione, D. Ziupa, M. Brunner, C. Bode, K. Odening (Freiburg im Breisgau) |
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Diskussion |
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(V106) |
Effects of cytoskeletal stabilization and disruption on the transverse tubular system |
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T. Seidel, M. Sonea, D. Fiegle, T. Baur, T. Volk (Erlangen) |
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Diskussion |
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(V107) |
Nanoscopic Fragmentation of the Calcium Release Unit Correlates with Hemodynamic Fate and Subtype of Valvular Aortic Stenosis |
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S. Brandenburg, H.-L. Schönberger, C. Lenz, T. Kohl, L. Otto, B. E. C. Beuthner, R. Topci, A. Vogelgesang, M. Puls, C. Jacobshagen, H. Urlaub, G. Hasenfuß, S. Lehnart (Göttingen) |
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Diskussion |
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(V108) |
Direct monitoring of cAMP in human cardiac myocytes reveals altered beta-adrenergic sensitivity and phosphodiesterase contributions in different diseases |
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F. Berisha, D. Westermann, B. Reiter, M. Schwarzl, B. Schrage, H. Reichenspurner, A. Bernhardt, S. Blankenberg, V. Nikolaev, C. Espinosa Molina (Hamburg) |
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Diskussion |
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(V109) |
The Effects of a Natural Light-gated Chloride-Channel in Cardiac Cells |
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R. Kopton, J. S. Baillie, S. A. Rafferty, R. Moss, C. Johnston, S. V. Prykhozhij, M. R. Stoyek, F. Smith, P. Kohl, T. A. Quinn, F. Schneider-Warme (Freiburg im Breisgau; Halifax, CA) |
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Diskussion |
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(V110) |
Acute β-adrenergic activation potentiates cytosolic Ca2+ cycling, but attenuates contractility in autophagy-defective mouse hearts |
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S. Kraler, M. Abdellatif, S. Ljubojevic, S. Sedej (Graz, AT) |
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Diskussion |
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(V111) |
Soluble Guanylate Cyclase as Therapeutic Target in Heart Failure: Functional and Proteomic Dynamics in Response to sGC stimulation after Experimental Aortic Stenosis in Mice |
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A. Benkner, J. Rüdebusch, K. Grube, K. Klingel, E. Witt, V. M. Dhople, E. Hammer, S. B. Felix (Greifswald, Tübingen) |
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Diskussion |