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(V944) |
Cardiomyocyte-specific depletion of protein kinase B isoforms disturbs cardiac bioenergetics during development of heart failure in mice |
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A. Heinen, T. Stemmer, S. Gödecke, U. Flögel, K. Köhrer, A. Gödecke (Düsseldorf) |
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Diskussion |
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(V945) |
The heat shock protein HSPA4 mitigates pathological cardiomyocyte remodeling possibly via benign enhancement of autophagy |
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S. Gersch, M. El Kenani, E. Sadrach, G. Hasenfuß, S. Lutz, K. Toischer, B. Mohamed, für die Studiengruppe: DZHK (Göttingen) |
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Diskussion |
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(V946) |
Nitro oleic acids attenuates systolic heart failure in a murine model of hypertensive heart disease |
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D. Mehrkens, S. Geißen, M. Adam, M. Pekarova, A.-K. Schätzle, M. Mollenhauer, L. Kubala, G. Ambrozova, B. Freeman, A. Klinke, S. Baldus, V. Rudolph (Köln; Brno, CZ; Pittsburgh, US) |
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Diskussion |
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(V947) |
Targeting fatty acid oxidation by Acetyl-CoA Carboxylase 2 deletion in pathological hypertrophy |
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J. Ritterhoff, O. Villet, S. C. Kolwicz Jr, D. Shao, R. Tian (Seattle, WA, US) |
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Diskussion |
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(V948) |
High and low intrinsic exercise capacity under pressure overload – impact on survival and cardiac & mitochondrial function |
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C. Schenkl, M. Wildgrube, P. Ackermann, A. Schrepper, E. Heyne, M. Schwarzer, S. Britton, L. G. Koch, T. Doenst (Jena; Ann Arbor, US) |
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Diskussion |
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(V949) |
Activation of the GIP system attenuates pressure overload-induced cardiac remodeling in mice |
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F. Kahles, C. Halim, J. Möllmann, R. W. Mertens, I. Diepolder, S. Diebold, R. Stöhr, C. Lebherz, N. Marx, M. Lehrke (Aachen) |
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Diskussion |