|
(V944) |
Cardiomyocyte-specific depletion of protein kinase B isoforms disturbs cardiac bioenergetics during development of heart failure in mice |
|
|
A. Heinen, T. Stemmer, S. Gödecke, U. Flögel, K. Köhrer, A. Gödecke (Düsseldorf) |
|
|
Diskussion |
|
(V945) |
The heat shock protein HSPA4 mitigates pathological cardiomyocyte remodeling possibly via benign enhancement of autophagy |
|
|
S. Gersch, M. El Kenani, E. Sadrach, G. Hasenfuß, S. Lutz, K. Toischer, B. Mohamed, für die Studiengruppe: DZHK (Göttingen) |
|
|
Diskussion |
|
(V946) |
Nitro oleic acids attenuates systolic heart failure in a murine model of hypertensive heart disease |
|
|
D. Mehrkens, S. Geißen, M. Adam, M. Pekarova, A.-K. Schätzle, M. Mollenhauer, L. Kubala, G. Ambrozova, B. Freeman, A. Klinke, S. Baldus, V. Rudolph (Köln; Brno, CZ; Pittsburgh, US) |
|
|
Diskussion |
|
(V947) |
Targeting fatty acid oxidation by Acetyl-CoA Carboxylase 2 deletion in pathological hypertrophy |
|
|
J. Ritterhoff, O. Villet, S. C. Kolwicz Jr, D. Shao, R. Tian (Seattle, WA, US) |
|
|
Diskussion |
|
(V948) |
High and low intrinsic exercise capacity under pressure overload – impact on survival and cardiac & mitochondrial function |
|
|
C. Schenkl, M. Wildgrube, P. Ackermann, A. Schrepper, E. Heyne, M. Schwarzer, S. Britton, L. G. Koch, T. Doenst (Jena; Ann Arbor, US) |
|
|
Diskussion |
|
(V949) |
Activation of the GIP system attenuates pressure overload-induced cardiac remodeling in mice |
|
|
F. Kahles, C. Halim, J. Möllmann, R. W. Mertens, I. Diepolder, S. Diebold, R. Stöhr, C. Lebherz, N. Marx, M. Lehrke (Aachen) |
|
|
Diskussion |