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(BS58) |
Loss of the Ectodomain Sheddase ADAM10 leads to decreased heart function, premature mortality and increased arrhythmia susceptibility |
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S. Weber, S. Meyer-Roxlau, S. Lämmle, M. Wagner, K. Lorenz, A. El-Armouche (Dresden, Dortmund) |
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(BS59) |
Novel insights into regulation and induction of proarrhythmic persistent Na current in human and mouse cardiomyocytes |
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P. Tirilomis, S. Ahmad, P. R. F. Bengel, N. Dybkova, L. S. Maier, T. H. Fischer, G. Hasenfuß, S. T. Sossalla (Göttingen, Regensburg) |
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(BS60) |
Proarrhythmic late sodium current and diastolic Ca2+-leak are regulated by sodium channel isoform NaV1.8 in human cardiac hypertrophy and mouse ventricular cardiomyocytes |
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P. R. F. Bengel, S. Ahmad, P. Tirilomis, N. Dybkova, L. S. Maier, T. H. Fischer, G. Hasenfuß, S. T. Sossalla (Göttingen, Regensburg) |
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(BS61) |
Role of sodium channel NaV1.8 in cellular pro-arrhythmias in human atrial cardiomyocytes |
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S. Ahmad, P. Tirilomis, N. Hartmann, N. Dybkova, T. H. Fischer, K. Streckfuß-Bömeke, N. Frey, G. Hasenfuß, L. S. Maier, S. T. Sossalla (Regensburg, Göttingen, Kiel) |
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(BS62) |
Increased Occurrence of Ventricular Arrhythmias in Type 2 Diabetic Mice |
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C. Jungen, P. Rajendran, S. Willems, C. Meyer, J. Ardell, J. Tompkins (Hamburg; Los Angeles, US) |
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(BS63) |
A novel SCN5A mutation with loss-of-function of NaV1.5 unmasked at 37 °C experimental temperature in a family with symptomatic Brugada syndrome |
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J. Müller-Leisse, K. Sanner, D. Duncker, T. König, C. Zormpas, R. Michalski, A. Leffler, C. G. Veltmann (Hannover) |