| (BS29) |
The Toll-like receptor 4 ligands S100A8 and S100A9 are crucial factors in viral cardiomyopathy |
|
M. Becher, D. Westermann, D. Lindner, T. Vogl, J. Roth, H.-P. Schultheiss, C. Tschöpe (Berlin, Münster) |
| (BS30) |
Changes in Renal Perfusion and Blood Pressure During Obstructive Apneas: Investigations in Spontaneously Breathing Anesthetized Pigs |
|
D. Linz, M. Löhn, W. Linz, H.-R. Neuberger, M. Böhm, K. Wirth (Homburg/Saar, Frankfurt am Main) |
| (BS31) |
The NDPK isoforms B and C are required for heart function and angiogenesis in the zebrafish |
|
N. Wolf, I. Abu-Taha, H.-J. Hippe, S. Lutz, B. Meder, H. A. Katus, S. Just, W. Rottbauer, T. Wieland (Heidelberg, Mannheim, Münster, Göttingen, Ulm) |
| (BS32) |
Directed Formation of Bioengineered Human Myocardium (BHM) |
|
J. Hudson, M. Tiburcy, W.-H. Zimmermann (Göttingen) |
| (BS33) |
Engineered Heart Tissue – A Useful Tool to Investigate Fast Epigenetic Dynamics in Cardiomyocytes |
|
L. M. Bartholdt, J. Stenzig, M. N. Hirt, A. Eder, A. Hansen, T. Eschenhagen (Hamburg) |
| (BS34) |
Role of minor and major histocompatibility genes in an allogeneic model of parthenogetic stem cell transplantation |
|
V. Muppala, M. Didie, S. Galla, L. Elsner, R. Dressel, W.-H. Zimmermann (Göttingen) |
| (BS35) |
Novel electrophysiological properties of dronedarone: Inhibition of human cardiac two-pore-domain potassium (K2P) channels |
|
C. Schmidt, F. Wiedmann, R. Becker, H. A. Katus, D. Thomas (Heidelberg) |
| (BS36) |
Cardiac Remodeling and Myocardial Dysfunction in Obese Spontaneously Hypertensive Rats |
|
D. Linz, M. Hohl, F. Mahfoud, J.-C. Reil, W. Linz, H.-P. Juretschke, T. Huebschle, H.-R. Neuberger, C. Neumann-Haefelin, H. Ruetten, M. Böhm (Homburg/Saar, Frankfurt am Main) |
| (BS37) |
ß-adrenergic stimulation in cardiac myocytes stimulates neuronal NOS-related NO production, but this NO does not control mitochondrial respiration |
|
M. Kohlhaas, S. Bergem, A. Nickel, M. Meiser, M. Hohl, U. Laufs, M. Böhm, C. Maack (Homburg/Saar) |
| (BS38) |
Reverse mode of the mitochondrial transhydrogenase consumes NADPH during cardiac workload and provokes ROS generation and maladaptive remodelling |
|
A. Nickel, J. R. Löffler, J.-C. Reil, A. von Hardenberg, M. Kohlhaas, M. Hohl, A. Kazakov, B. Pasieka, I. Bogeski, R. Kappl, M. Hoth, M. Böhm, C. Maack (Homburg/Saar) |
| (BS39) |
cAMP-induced nuclear accumulation of HDAC5 protects against cardiomyocyte hypertrophy |
|
T. He, L. Chen, G. Han, C. Heft, H. A. Katus, J. Backs (Heidelberg) |