| P117 | Predominant left atrial antiarrhythmic actions of Ikur-, Ikr-, Iks-, IkAch-, betablockers, and class Ic-drugs in pigs. |
| 1K.Knobloch, 2J.Brendel, 2A.E.Busch, 2M.Bleich, 2K.J.Wirth | |
| 1Thorax-, Herz- & Gefäßchirurgie, Medizinische Hochschule, Hannover; 2Aventis Pharma Germany, DG Cardiovascular Diseases, Frankfurt am Main. | |
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Left (LA) and right atrial (RA) side differences of refractoriness as well as distinct pulmonary veins are reported in different species. The effects of novel atrial selective Ikur-blockers were investigated in this study in contrast to IKr-blockers dofetilide (DO), azimilide (AZ), ibutilide (IB), amiodarone (AM), IKs-blocker HMR 1556, IKAch-blocker atropine (ATR), class Ic drugs flecainide (FL) and propafenone (PR), and βblockers d,l-sotalol (SO), atenolol (ATE), and esmolol (ES) on LA and RA and ventricular refractoriness, and left atrial vulnerability (LAV)in vivo in pigs. Methods: In pentobarbital-anesthetized pigs (n=81) LA and RA effective refractory periods (ERP) were measured with the S1-S2-extrastimulus-method. LAV was assessed after a S2-extrastimulus applied on the LA. Results: All IKur-blockers, AVE 0118 (1mg/kg), S9947 (3mg/kg) and S20951 (3mg/kg) prolonged left stronger than right atrial refractoriness and did not change QTc-time. All IKr-blockers prolonged predominantly right vs. left atrial ERP as did IKs-blocker HMR 1556. AM was equally effective on both atria. IKAch-blocker atropine prolonged only left atrial ERP, as did pure βblockers with predominant left atrial ERP prolongation and class Ic drugs, FL and PR, while d,l-sotalol acted predominantly right atrial. IKur-blockers, S9947 and S20951, ibutilide, and d,l-sotalol significantly decreased left atrial vulnerability (-100%, -82%, -53%, -53%, p<0.05). Conclusions: IKur-blockers, AVE 0118, S9947 and S20951, exhibit stronger left atrial effects with no effect on ventricular repolarization. By contrast, IKr-blockers, IKs-blockers, and d,l-sotalol exert predominant right atrial and known ventricular effects. These data demonstrate atrial side differences of antiarrhythmic drugs. IKur-blockers inhibit atrial tachyarrhythmias stronger than all available drugs, therefore IKur-blockers seem to be promising new atrial selective antiarrhythmic drugs. |