79. Jahrestagung der Deutschen Gesellschaft für Kardiologie

Samstag, 6. April 2013 (Saal 9) 08:30-10:00

Freie Vorträge

Neue Mechanismen der kardialen Fibrose

Vorsitz: R. Hambrecht (Bremen), A. Remppis (Bad Bevensen)

(V1543) Myofibroblasts as inflammatory supporter cells trigger cardiac inflammation in heart failure due to increased mechanical stretch

D. Westermann, D. Lindner, H.-P. Schultheiss, C. Tschöpe (Berlin)
(V1544) MiR-21 mediates interstitial fibrosis in cardiac allografts by supporting monocyte to fibroblast transition

S. K. Gupta, J. Fiedler, C. Falk, S. Heymans, T. Thum (Hannover; Maastricht, NL)
(V1545) The deficiency of PAR2 leads to an age dependent cardiac fibrosis and left ventricular dysfunction

A. Weithäuser, R. Malz, K. Savvatis, H.-P. Schultheiss, U. Rauch-Kröhnert (Berlin)
(V1546) The role of stabilin-1 as a marker for alternative macrophage activation in development of fibrosis in myocardium of dilatative cardiomyopathy patients with left ventricular assist device implantation

K. Wassilew, E. Potapov, C. Schmuttermaier, A. Mickley, A. Gratchev, R. Hetzer, J. Kzhyshkowska (Berlin, Mannheim)
(V1547) Inhibition of the Overactivated Cannabinoid-1 Receptor Signaling Attenuates Cardiac Dysfunction, Inflammation, and Fibrosis in Diabetic Cardiomyopathy

S. Batkai, R. Mohanraj, P. Mukhopadhyay, B. Horvath, R. Cinar, G. Hasko, K. Mackie, L. Liaudet, P. Pacher (Hannover, Lausanne; Bethesda, Newark, Bloomington, US)
(V1548) Cardiac-specific deletion of the CaMKII isoforms δ and γ protects against angiotensin-dependent perivascular fibrosis

S. Schmidt, M. Kreußer, L. H. Lehmann, J. Krebs, H. A. Katus, J. Backs (Heidelberg)